Titlebook: Mechanisms and Consequences of Proton Transport; Tetsuro Urushidani,John G. Forte,George Sachs Book 2002 Springer Science+Business Media N

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Structure-Activity Relationships of Na,K-ATPase and H,K-ATPase likely that the binding sites for ions and for certain inhibitory drugs are located within or close to transmembrane segments. By making chimeras of the α-subunits of Na,K-ATPase and H,K-ATPase, information could be obtained about transmembrane segments that could be responsible for the inhibitor and cation specificity.

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HCl Causes Less Intracellular Acidification In Surface Epithelial Cells Of Isolated Necturus Gastrictly larger intracellular acidification than exposure to HCl. The degree of acidification was not dependent on the valence or nature of the anionic counterion of the acid, but significantly correlated with the amount of undissociated molecular form of the acid in the solution.

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Induction of Small Intestinal Damage by Inhibition of Both NO Synthase and COX-2inhibition of COX-1 was accompanied by the mucosal expression of COX-2 (.), it is assumed that PGs produced by COX-2 contribute to the mucosal integrity by antagonizing deleterious events caused by inhibition of COX-1.

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Book 2002 gastric acid secretion. These include development of the H2-receptor blockers, defining the signaling pathways for the regulation of acid secretion, identifying the gastric proton pump, discovery and development of proton pump inhibitory drugs, and elucidating the physiology and biochemistry of Helicobacter pylori.

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