Titlebook: Gastric Cytoprotection; A Clinician’s Guide Daniel Hollander,Andrzej S. Tarnawski Book 1989 Springer Science+Business Media New York 1989 a

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https://doi.org/10.1007/978-1-4684-5697-4attention; cell; gastrointestinal tract; muscle; pathogenesis; pathophysiology; physiology; production; regu

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C. W. Lewis,J. G. Anderson,J. E. Smiths been anastomosed to the stomach. Peptic ulcers have the common attribute of occurring only in those parts of the alimentary tract which are exposed to gastric secretions. This feature, together with the demonstration that gastric juice has the capacity to destroy animal tissues, has resulted in th

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Eric D. Spitzer,George S. Kobayashi occupies a relatively small area at the esophagogastric junction contains mucoid glands with some acinar arrangement. The mucosa of the fundic area and body (corpus) of the stomach consists of oxyntic glands draining into gastric pits (Fig. 2). Oxyntic glands consist of parietal cells secreting aci

Fecal-Impaction

https://doi.org/10.1007/978-94-6091-633-5es on the mechanisms of gastrointestinal (GI) mucosal damage are frequently applicable to the understanding of mucosal protection. One of the key aspects of these extrapolations is that, according to conclusions derived during the last decade of research on mucosal injury and protection, both proces

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https://doi.org/10.1007/978-1-4757-9293-5ghout the GI tract because it maintains the functional integrity of the epithelium and is also necessary for the repair of mucosal injury. Thus, it is reasonable to expect that certain conditions and agents that predispose to peptic ulcer disease or mucosal injury may do so in part by affecting epit

Agnosia

Ryan P. Rodgers,Alan G. Marshallo resists all but the harshest of miscellaneous chemical insults. It is no surprise, then, that this tissue has intrigued doctors and scientists for centuries, particularly with regard to how it achieves such remarkable Teflon-like chemical resistance properties. It is only relatively recently, howe