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Titlebook: Eicosanoids and other Bioactive Lipids in Cancer, Inflammation, and Radiation Injury 3; Kenneth V. Honn,Lawrence J. Marnett,Patrick Y.-K.

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书目名称Eicosanoids and other Bioactive Lipids in Cancer, Inflammation, and Radiation Injury 3
编辑Kenneth V. Honn,Lawrence J. Marnett,Patrick Y.-K.
视频video
丛书名称Advances in Experimental Medicine and Biology
图书封面Titlebook: Eicosanoids and other Bioactive Lipids in Cancer, Inflammation, and Radiation Injury 3;  Kenneth V. Honn,Lawrence J. Marnett,Patrick Y.-K.
出版日期Book 1997
关键词apoptosis; cancer; cell; lymphocytes
版次1
doihttps://doi.org/10.1007/978-1-4899-1813-0
isbn_softcover978-1-4899-1815-4
isbn_ebook978-1-4899-1813-0Series ISSN 0065-2598 Series E-ISSN 2214-8019
issn_series 0065-2598
copyrightThe Editor(s) (if applicable) and The Author(s), under exclusive license to Springer Science+Busines
The information of publication is updating

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Wendelyn H. Inman,Graham Carpenter vitronectin, thrombospondin, and von Willebrand factor.. Cells expressing these receptors have an advantage to adhere to a variety of basement membrane proteins and thereby generate different matrix driven cellular responses.
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Human and Rat Cyclooxygenases are Pharmacologically Distinctmpounds. The active compounds are subsequently evaluated mostly using rat or mouse in vivo models. Unless the COX enzymes used for the in vitro screening is pharmacologically identical to the COX enzyme of the in vivo model, the in vivo results may be misleading.
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Effects of Secretory PLA2 on Rat Peritoneal Mast Cells Activated by Different Secretagoguesis enzyme in non-digestive organs suggested that it may play a role in cell proliferation and in smooth muscle contraction.. The concentration of sPLA.-II is increased in synovial fluids and plasma of patients with diverse inflammatory diseases.. The sPLA.-II may hence play important roles in the pathogenesis of these inflammatory diseases.
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Function of Calcium-Independent Phospholipase A2 in Arachidonic Acid Metabolism in P388D1 Macrophagetely 14 kDa), the structurally unrelated Ca.-dependent, high molecular mass enzymes (85 kDa), and the recently described Ca.-independent enzymes.. All three families have been implicated in AA release in a variety of cells, their relative contribution being dependent on the agonist and cell type considered.
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