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Titlebook: Clinical Experiences with Budipine in Parkinson Therapy; Franz Gerstenbrand,Werner Poewe,Gerald Stern Conference proceedings 1985 Springer

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PICKT: A Solution for Big Data Analysisderangement of the relationship of the neurotransmitters themselves (Barbeau 1962; McGeer et al. 1961; Siegfrid 1976). The nigrostriatal dopaminergic system is certainly involved in the pathogenesis of tremor, even if a clear relationship between tremor and the damage of the dopaminergic system does not exist (Rinne et al. 1982).
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Rough Sets and Knowledge Technologytalepsy have been shown in animal experiments suggesting a dopaminergic mode (Brand and Menge 1982; see the chapter by Menge and Brand) of action. On the other hand, weak anticholinergic properties have been demonstrated also (see the chapter by Menge and Brand).
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,Dopaminergic Agonists and Receptors: Their Relevance to the Treatment of Parkinson’s Disease,rbidopa and benserazide) that blocked the conversion of levodopa to dopamine by L-aromatic amino acid decarboxylase. These enzyme inhibitors were chosen because they did not readily cross the blood-brain barrier, so they did not impede the formation of dopamine in the striatum.
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,Controlled Single-Blind Trial of Budipine, L-Dopa, and Amantadine in 102 Patients with Parkinson’s hardly afford to ignore the patient’s own feeling, be it ever so transient and subjective. Above all, doctors are supposed primarily to take patients’ well-being into consideration, being concerned additionally with the results obtained with sophisticated measurements.
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Granular Computing in Multi-agent Systemse nucleus caudatus and also in the whole brain. Evidence that this increase was brought about monamine oxidase (MAO) or catechol Omethyltransferase (COMT) inhibition could not be found. Rather it is suggested that the increase is due to a reuptake inhibition.
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