Titlebook: Clinical Experiences with Budipine in Parkinson Therapy; Franz Gerstenbrand,Werner Poewe,Gerald Stern Conference proceedings 1985 Springer

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Granular Computing in Multi-agent Systemss found that budipine: (a) is an active antiparkinsonian drug, especially against parkinsonian tremor (also against oxotremorine-induced tremors in laboratory animals); (b) enhances the effectiveness of L-dopa treatment in parkinsonism; (c) produces mood elevation; and (d) exhibits a peripheral symp

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Granular Computing in Multi-agent Systemsic adjuvant in Parkinsons’s disease. In reserpine-induced Parkinson syndrome, budipine brings about an increase in the levels of biogenic amines in the nucleus caudatus and also in the whole brain. Evidence that this increase was brought about monamine oxidase (MAO) or catechol Omethyltransferase (C

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A New Discernibility Matrix and Functiontions for treating patients (Siegfried 1982) suffering from parkinsonian syndrome. The present report includes a summary of the studies hitherto conducted on the pharmacokinetics and metabolism of budipine.

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Peng Wen,Yuefeng Li,Guoyin Wangtenbrand et al. 1963; Cotzias et al. 1967). In recent years, there has been growing awareness of the shortcomings of this form of therapy, characterized by declining clinical efficacy on the one hand and increase in central side effects on the other during long-term levodopa treatment (Fahn and Caln

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Rule-Based Estimation of Attribute Relevancecularly life partners, or their general practitioners. An experienced physician, e.g., consultant neurologist, will draw comparisons with numerous parkinsonian patients in addition to his clinical observations and measurements of the patient in question. His sound and critical judgment, however, can

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