Titlebook: Chronic Myelocytic Leukemia and Interferon; Pathophysiological, Dieter Huhn,Klaus Peter Hellriegel,Norbert Niederl Conference proceedings

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https://doi.org/10.1007/978-3-662-29788-9astic-transformed stem cell [10]. In more than 90% of cases, an indicative reciprocal translocation t(9.22), known as the “Philadelphia chromosome,” is present [39]. The observation that natural interferon-alpha (IFN-α) and fibroblast interferon (IFN-β) caused inhibitory effects on the in vitro grow

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,Konzeption und Beschreibung der Geräte,nitor cells. The best-studied of these precursor cells are the stem cell-like progenitors (CFU-GEMM), the early erythroid progenitors (BFU-E), the granulocytic progenitors (CFU-C), and the megakaryocytic progenitors (CFU-M) [2, 4]. The levels of these colony forming cells reflect the functional capa

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Perspektiven der Mathematikdidaktik 20, 28, 35, 41]. CML has remained uniformly fatal, and the only curative therapy available up to now has been bone marrow transplantation [16, 58]. This, however, can be offered only to the small proportion of CML patients of sufficiently young age who have an HLA-compatible donor.

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https://doi.org/10.1007/978-94-009-2415-4 50 years. Many forms of treatment, such as busulfan, hydroxyurea, dibromomannitol, melphalan, mercaptopurine, thioguanine, and splenic irradiation, are effective in controlling the manifestations of CGL during the chronic phase, but none of these agents delays the onset of the blast phase or prolon

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,Treatment of Chronic and Acute Phase Chronic Myelogenous Leukemia with Interferon-α2b and, InterferThe interferons (IFNs) are a group of proteins which can exert a wide range of biologic effects on normal tissue cells, host immune defense cells, and cancer cells [5, 9,12, 20]. With regard to their immunochemical properties and their cellular origin, three types of IFNs have been distinguished: α, β, and γ [7].