Titlebook: Chronic Myelocytic Leukemia and Interferon; Pathophysiological, Dieter Huhn,Klaus Peter Hellriegel,Norbert Niederl Conference proceedings

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In Vitro Effects of Recombinant Human Tumor Necrosis Factor Alpha and Interferon Gamma on Normal anthe availability of very pure recombinant material that has now been used in several clinical trials to explore its in vivo activity against various solid tumors ([11] R. Mertelsmann, H. Gamm, F. Herrmann, to be published). TNF-α is now expected to be included in the clinical setting of hematological malignancies.

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Cytogenetic Findings in Myeloproliferative Disorders,uring the last decade. The rise of tumor cytogenetics was caused by the development of refined chromosome banding techniques which allowed the detection of chromosomal rearrangements that had previously been overlooked. With the improvement of these techniques, chromosome anomalies can now be demons

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Moleculargenetic Analyses of Chronic Myelocytic Leukemia, in some members of a group of cellular genes, calles “proto-oncogenes” [32, 44]. Some 40 different oncogenes have been identified so far. The few available data indicate that this heterogenous set of genes plays a crucial role in the regulation of cell proliferation and differentiation. Various mol

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Molecular Biological Investigation in Chronic Myelogenous Leukemia Patients Undergoing Interferon T22 as result of a translocation t(9:22)(q 34; q 11). By this translocation the cellular proto-oncogene c-abl is transferred from chromosome 9 to chromosome 22 and inserted into the so-called breakpoint cluster region (.). This region is part of a large (~ 100 kb) . gene, the function of which is yet

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