Titlebook: Checkpoint Controls and Cancer; Volume 2: Activation Axel H. Schönthal Book 2004 Humana Press 2004

尽忠

iFTS Dynamic Management Views and Functions,es and spindle tension, chromosome positioning, and kinetochore signaling by the Mad2 or Bub1 checkpoint proteins. We also describe a bi-parameter flow cytometric assay, using either MPM-2 or anti-phospho-(Ser10)-histone H3 antibodies, for quantitating mitotic cells.

轻推

束以马具

ARK

Introduction to Flex Applications,f pRb by cyclin-dependent kinases (Cdks) has been studied extensively, the role(s) of protein phosphatase 1 (PP1) in controlling pRb are only partially understood. In this chapter, we will describe experimental approaches to investigate the interactions between pRb and PP1. Methods will be presented

RECUR

Project Planning for Flex and Spring,y mechanisms. Here we present a strategy to identify genes regulated by specific transcription factors in the human genome, and apply it to p53. We first collected promoters or introns of all genes available using two methods: GenBank annotation and a computationally derived transcript map. The “Fin

粗糙滥制

observatory

Building the Flex User Interface,activated in response to DNA damage. In addition, p21 interacts directly with proliferating cell nuclear antigen (PCNA), thereby inhibiting DNA replication. More controversial is the role of p21 in DNA repair, since both inhibition of and requirement for nucleotide excision repair have been suggeste

催眠

Flex and Spring Integration Architecture,pment, progression, and resistance to treatment. Whereas semi-quantitative methods, such as Northern blotting analysis, allow only a dichotomous differentiation between positive and negative gene expressions, the realtime quantitative polymerase chain reaction (qRT-PCR) combines a large range of res

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态学