Titlebook: Antiviral Resistance in Plants; Methods and Protocol John M. Watson,Ming-Bo Wang Book 2012 Springer Science+Business Media, LLC 2012 Antivi

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Peter Kuhn,Matthias Buchinger,Dian Baltare it was called post-transcriptional gene silencing, PTGS) RNAi is a gene down-regulation mechanism since demonstrated to exist in all eukaryotes. In RNAi, small RNAs (of about 21–24 nucleotides) function to guide specific effector proteins (members of the Argonaute protein family) to a target nucl

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Hans J. Scholl,Eric E. Holdemane molecular immune mechanism against invading viruses. The antiviral RNAi pathway is initiated with the generation of virus-derived short-interfering RNAs (siRNAs) that are used for subsequent sequence-specific recognition and degradation of the cognate viral RNA molecules. As an efficient counter-d

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Peter Kuhn,Matthias Buchinger,Dian Baltaus that has had its genome modified to include a sequence from the host gene to be silenced. DNAβ and DNA1 are satellite and single-stranded DNA molecules associated with begomoviruses (family .). We converted DNAβ and DNA1 into gene-silencing vectors. The VIGS vectors can induce silencing efficient

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Hans J. Scholl,Eric E. Holdemanmage caused by a severe challenge strain/isolate of the same virus. Mild strain cross protection (MSCP) has been used extensively to control losses caused by a few major virus diseases in some parts of the world. So far, none of the many proposed mechanisms can fully explain the intact process of MS

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Stijn Wouters,Marijn Janssen,Joep Crompvoetsrigger the host plant’s RNA-silencing machinery and ultimately become the target of RNA silencing. PTGS is, therefore, an attractive endogenous process that can be exploited to study gene function. One of the most efficient approaches of initiating PTGS is through virus-induced gene silencing (VIGS)

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