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Titlebook: Advances in Research on Neurodegeneration; Volume 7 Y. Mizuno,D. B. Calne,M. B. H. Youdim Conference proceedings 2000 Springer-Verlag Wien

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Nanostructure Science and Technologysible for the pathogenesis of autosomal recessive juvenile parkinsonism (AR-JP). Various mutations were found in AR-JP patients of Japanese and other ethnic origins, providing a definitive evidence for the Parkin to be a causative gene for AR-JP. The predicted structure of Parkin protein and its mut
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https://doi.org/10.1007/978-0-387-74778-1sis, cell turn over and removal of harmful cells. Disruption of the regulation of apoptosis may result in various diseases, including cancer and autoimmune diseases both associated with inhibition of apoptosis and various degenerative disorders associated with enhancement of apoptosis, and therefore
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Optimal Disturbances in Boundary Layers,production of Aβ and the development of the disease..Thus, Aβ may not be the sole active fragment of β-amyloid precursor protein (βAPP) in the neurotoxicity assiciated with AD..We focused on the amyloidegenic carboxyl terminal fragments of βAPP containing the full length of Aβ (CT.). We synthesized
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Statistical Analysis and Modeling of Data,us human motor disorders. This paper reviews evidence from various sources which suggests that this standard model only incompletely accounts for aspects of basal ganglia function, and thus requires modification.
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David T. Melnekoff,Alessandro Laganàase, the level of N-methyl(R)salsolinol increased in cerebrospinal fluid and the high activity of a synthesizing enzyme, (R)salsolinol Nmethyltransferase, was detected in lymphocytes. This isoquinoline induced apoptotic DNA damage in human dopaminergic neuroblastoma SH-SY5Y cells. Among catechol iso
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