Titlebook: Advances in Research on Neurodegeneration; Volume 7 Y. Mizuno,D. B. Calne,M. B. H. Youdim Conference proceedings 2000 Springer-Verlag Wien

cajole

aptitude

步履蹒跚

吊胃口

SpringerBriefs in Computer Sciencelutamine stretch. The prominent anticipation and broad spectrum in the clinical presentations of DRPLA have been demonstrated to be tightly correlated with the instability of CAG repeats in the DRPLA gene. Discovery of the causative gene for DRPLA has made it possible to investigate molecular mechan

FLAX

parsimony

https://doi.org/10.1007/978-0-387-68825-1cules are able to induce relatively selective injury of oligodendrocytes (OLs) and their myelin membranes. The selectivity of this injury could be conferred either by the properties of the effectors or the targets. The former would involve antigen specific recognition by either antibody or T cell re

hematuria

VERT

Statistical Analysis and Modeling of Data,us human motor disorders. This paper reviews evidence from various sources which suggests that this standard model only incompletely accounts for aspects of basal ganglia function, and thus requires modification.

GRUEL

Huntington disease: new insights on the role of huntingtin cleavage,ransgenic mice expressing normal (YAC18) and mutant (YAC46 and YAC72) human huntingtin in a developmentally appropriate and tissue-specific manner identical to the pattern of expression of endogenous huntingtin. YAC46 and YAC72 mice show early electrophysiological abnormalities indicating neuronal c

acrimony

PARKIN as a pathogenic gene for autosomal recessive juvenile parkinsonism,sible for the pathogenesis of autosomal recessive juvenile parkinsonism (AR-JP). Various mutations were found in AR-JP patients of Japanese and other ethnic origins, providing a definitive evidence for the Parkin to be a causative gene for AR-JP. The predicted structure of Parkin protein and its mut