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Titlebook: Advances in Research on Neurodegeneration; Volume 7 Y. Mizuno,D. B. Calne,M. B. H. Youdim Conference proceedings 2000 Springer-Verlag Wien

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期刊全称Advances in Research on Neurodegeneration
期刊简称Volume 7
影响因子2023Y. Mizuno,D. B. Calne,M. B. H. Youdim
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图书封面Titlebook: Advances in Research on Neurodegeneration; Volume 7 Y. Mizuno,D. B. Calne,M. B. H. Youdim Conference proceedings 2000 Springer-Verlag Wien
影响因子We organized the Seventh International Winter Conference on Neurodegeneration and Neuroinflammation in a small town of Karuizawa in Nagano Prefecture in Japan on January 20 to 22, 1999. Karuizawa is a nice summer as well as winter resort close to the place for winter Olympic game in the year of 1998. Over 40 scientists gathered together and made hot discussion. Neurodegeneration and neuroinflammation are two major and important neurologic disorders, in which satisfactory neuroprotective and neurorestorative treatment is not available yet. For this purpose, understanding of molecular mechanisms of neuronal cell death in these two disorders is imperative. Recently, pathologic processes common to these two groups of disorders have been identified such as the involvement of inflammatory changes, microglia, cytokines, and apoptosis. We intended to involve scientists working in these two major fields together to participate in hot and fruitful discussion for the understanding of neuronal death and for developing newer methods of treatment. The science is progressing so rapidly today and we are working in a very narrow specialized area. Sometimes, we are ignorant about important discoveri
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Computational Methods for Physicistshat continuous sustained stimulation of dopamine receptor may be responsible for its unusual pharmacological activity. Apomorphine has a far more broad neuroprotective activity in the various models as compared with 1-selegiline and may therefore be an ideal drug to study neuroprotection in parkinso
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https://doi.org/10.1007/978-0-387-68825-1of therapeutic delivery systems, which would reach the site of disease activity within the CNS, will permit the administration of inhibitors either of the cell death pathway or of effector target inter action and opens new avenues to neuroprotection approach.
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