Titlebook: Advances in Research on Neurodegeneration; 3 & 4 Y. Mizuno,M. B. H. Youdim (Faculty of Medicine),P. Conference proceedings 1997 Springer-Ve

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Central nervous system cytokines and their relevance for neurotoxicity and apoptosis,y, it is necessary to study the temporal and spatial expression of complex networks of cytokines, growth factors, neuropeptides, and neurotransmitters. This effort is currently in progress in many centers. Modulation of cytokine function in the central nervous system represents a new therapeutic strategy for neurodegeneration.

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Induction of experimental autoimmune encephalomyelitis by CD4+ T cells specific for an astrocyte pr T cells which induced an inflammatory response in the CNS and eye of naive syngeneic recipients. The distribution of lesions in this novel model of EAE resembles that seen in some patients with MS, suggesting that the initial autoimmune insult in MS may be directed against a non-myelin antigen co-expressed in the CNS and extra-neural tissues.

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Role of interferons in demyelinating disease,of administration, preceded this trial and provided the scientific foundation for its success. Beta interferon therapy probably inhibits gamma interferon to achieve its therapeutic effect. Future MS therapy may require combination treatment with multiple agents with complimentary immunologic effects.

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从属

Introduction and General Equationsith respect to the etiopathogenesis of this neurodegenerative disorder. As a possible primary abnormal event in late-onset sporadic DAT, a prolonged desensitization of the neuronal insulin receptor is assumed to be responsible for cascade-like abnormalities in oxidative energy metabolism and related metabolism with impacts on amyloid formation.

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J. Killeen,G. D. Kerbel,A. A. Mirins which increase the synthesis and release of DA reduce the symptoms, they also may serve to exacerbate the neurodegenerative process. We propose that the treatments which increase the antioxidant capacity of brain may be protective.

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