Titlebook: Advances in Research on Neurodegeneration; 3 & 4 Y. Mizuno,M. B. H. Youdim (Faculty of Medicine),P. Conference proceedings 1997 Springer-Ve

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书目名称Advances in Research on Neurodegeneration影响因子(影响力)




书目名称Advances in Research on Neurodegeneration影响因子(影响力)学科排名




书目名称Advances in Research on Neurodegeneration网络公开度




书目名称Advances in Research on Neurodegeneration网络公开度学科排名




书目名称Advances in Research on Neurodegeneration被引频次




书目名称Advances in Research on Neurodegeneration被引频次学科排名




书目名称Advances in Research on Neurodegeneration年度引用




书目名称Advances in Research on Neurodegeneration年度引用学科排名




书目名称Advances in Research on Neurodegeneration读者反馈




书目名称Advances in Research on Neurodegeneration读者反馈学科排名

insidious

,Models of Alzheimer’s disease: cellular and molecular aspects,ion. While Cortisol and insulin have opposing effects on glucose metabolism, desensitization of the neuronal insulin receptor results in metabolic abnormalities. In the normal aging brain, glucose/energy metabolism is decreased slightly. In the majority of cases, Alzheimer’s disease is sporadic and

abracadabra

,The relationship of Alzheimer-type pathology to dementia in Parkinson’s disease,ility to dementia in patients with PD. Cortical pathologies of Alzheimer and Lewy body type seem to be the major determinants of dementia. The prevalence of Alzheimer’s disease is not increased in PD, but “early” cortical Alzheimer lesions (usually sub-clinical in normal controls) are frequently ass

Guileless

Models to study the role of neurotrophic factors in neurodegeneration,rons during early development has in many cases been well established. Recent studies have shown that neurotrophic factors can also protect vulnerable neurons against a variety of mechanical and chemical injuries. The role and the effects of neurotrophic factors in various neurological diseases are

inquisitive

记忆法

Immunological aspects of experimental allergic encephalomyelitis and multiple sclerosis and their aathological studies suggest, however, that T lymphocytes directed against myelin antigens are involved in the pathogenesis of MS. The examination of an experimental animal model for MS, experimental allergic encephalomyelitis (EAE), demonstrated that myelin basic protein- (MBP) or proteol-ipidprotei

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抱怨

Animal model and in vitro studies of anti neurofilament antibodies mediated neurodegeneration in Alilament protein NF-H of cholinergic neurons. Prolonged immunization of rats with these molecules but not with other NF-H isoforms results in cognitive impairments. This animal model, termed experimental autoimmune dementia (EAD), supports a role for such antibodies in neurodegeneration in AD..In the

Arctic

正论