Titlebook: Oxygen Radicals in the Pathophysiology of Heart Disease; Pawan K. Singal Book 1988 Kluwer Academic Publishers 1988 cardiovascular.cardiova

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The Role of Lipid Peroxidation in the Myocardium in Stress and Antioxidant Protection of the Heart,endogenous factor like myocardial infarction (4,5) involves the activation of lipid peroxidation (LP) in the myocardium. Preliminary administration of antioxidants limits or eliminates the LP activation (6,7,8) and at the same time decreases or completely eliminates diturbances of the heart metaboli

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Irwin Fridovichtsvermögen eines bestimmten Magneten bei Raumtemperatur = Wicklungstemperatur (22°C) und bei Betrieb mit Nennerregerspannung wieder, wenn der volle Hub des Magneten ausgenutzt wird. Zur Betätigung eines Wegeventils wird jedoch nur ein bestimmter Arbeitsbereich ausgenutzt, der von der benötigten Steu

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Role of Oxy-Radicals in Postischemic Myocardial Dysfunction,ugh early reperfusion limits infarct size, this beneficial effect does not lead to immediate functional improvement; rather, the return of contractility in tissue salvaged by reperfusion is delayed for hours, days or even weeks (1–9), a phenomenon that has been termed “stunned myocardium” (10).

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Book 1988his discovery, it was found that oxygen was both a life-sustaining and life threatening inhalant as it plays a role in the two extremes of the animal kingdom: life and death. In the subse­ quent years, we have made major strides in understanding the role of oxygen in maintaining life and volumes of

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Oxygen Radical Mediated Protein Oxidation in Heart,(8). Superoxide can be reduced further to other reactive oxygen metabolites. The dismutation of superoxide by superoxide dismutase produces hydrogen peroxide (H.O.) which, in the presence of metal catalysts, can form the hydroxyl free radical (OH·) by the Fenton, or Haber-Weiss pathways (9).