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Titlebook: Oxygen Radicals in the Pathophysiology of Heart Disease; Pawan K. Singal Book 1988 Kluwer Academic Publishers 1988 cardiovascular.cardiova

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书目名称Oxygen Radicals in the Pathophysiology of Heart Disease
编辑Pawan K. Singal
视频video
丛书名称Developments in Cardiovascular Medicine
图书封面Titlebook: Oxygen Radicals in the Pathophysiology of Heart Disease;  Pawan K. Singal Book 1988 Kluwer Academic Publishers 1988 cardiovascular.cardiova
描述Over two centuries ago, oxygen was discovered as "air vital": the component of the earth‘s atmosphere necessary for life. Less than five years after this discovery, it was found that oxygen was both a life-sustaining and life threatening inhalant as it plays a role in the two extremes of the animal kingdom: life and death. In the subse­ quent years, we have made major strides in understanding the role of oxygen in maintaining life and volumes of information are now available on this topic. Our knowledge of the contribution of oxygen in cellular dysfunction and cell death which for the most part had lagged behind has begun to catch up. The deleterious ef­ fects of oxygen radicals and activated oxygen species on a variety of biological systems have now been described. Recently attention has also been focused on the toxic effects of oxygen on the cardiovascular system. The major aim of the present treatise is to offer an integrated view of the pathophysiological aspects of oxygen toxicity in the heart and blood vessels coupled with a review of therapeutic approaches (hopes?) with free radical scavengers and antioxidants. Internationally known expert investigators provide a concise and
出版日期Book 1988
关键词cardiovascular; cardiovascular system; cell; heart; lipide; metabolism; pathophysiology; physiology; researc
版次1
doihttps://doi.org/10.1007/978-1-4613-1743-2
isbn_softcover978-1-4612-8979-1
isbn_ebook978-1-4613-1743-2Series ISSN 0166-9842
issn_series 0166-9842
copyrightKluwer Academic Publishers 1988
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Xanthine Oxidase is not Likely to be a Source of Injurious Free Radicals in the Ischemic Human Hearse NAD+ as the electron acceptor during the oxidation of purines (6,7). Thirty minutes of ischemia in the dog heart caused about 30% of the enzyme to be converted to the oxidase form (3) which can utilize molecular oxygen as an electron acceptor, reducing that oxygen to Superoxide and hydrogen perox
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