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Titlebook: Lessons from Animal Diabetes VI; 75th Anniversary of Eleazar Shafrir Book 1996 Birkhäuser Boston 1996 Diabetes.Diabetes mellitus.genetics.

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Mitsuo Itakura M.D., Ph.D.,Setsuko II,Mika Ohta,Takashi Yamaokagh inspection cost. Visual inspection for pressure vessels can be done by an Unmanned Aerial Vehicle (UAV) with the sensing module. Image enhancement techniques and image processing techniques are vital in the UAV inspection of pressure vessels. However, there are several issues to be overcome in th
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Stephen C. Blair,Ian D. Caterson,Gregory J. Cooneyhere (see Chaps. 5 and 6). A more direct approach is to search for favorable conditions and traces of life on other celestial bodies, both in the solar system and beyond. Clearly, there is little chance of encountering nonhuman intelligent beings in the solar system. But there could well be primitiv
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Barbara J. Frankel consequence of stellar evolution, the present cosmic abundances of chemical elements came about. Planets do not form independently; rather, their generation is an inevitable byproduct of star formation. This is because the gravitational collapse of large amounts of gas and dust from the interstella
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Expression of glutamic acid decarboxylase (GAD) and its role as an autoantigen in insulin-dependent models of human IDDM, the non-obese diabetic (NOD) mouse and the Bio-Breeding (BB) rat.. Similar pathogenic mechanisms are thought to result in human IDDM.. In all three species, autoimmune B-cell destruction appears to be a highly selective process, with relative sparing of the other islet cell typ
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Molecular genetics of insulin-dependent diabetes mellitus: Analysis of congenic strains individuals. Both in animal models and humans, major genetic susceptibility to IDDM has been mapped to the major histocompatibility complex (MHC). In addition, recent genome screening has mapped non-MHC susceptibility genes to chromosomal segments in the mouse and human genomes. For most loci, howe
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Role of macrophages and cytokines in B-cell deathor cells of autoimmunity, or as effector cells of B-cell destruction. In the spontaneously diabetic NOD mouse, macrophages and dendritic cells are the first cells infiltrating at perivascular and peri-insular regions in early insulitis beginning at the age of 3 wk.. These cells start to invade the i
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Immunotherapy of insulin-dependent diabetes mellitus: Tolerance after short-term anti—;IL-2 receptorted. and occurs in all mammals.. Investigations of animal strains predominantly used in diabetes research, the NOD mouse and the BB rat, demonstrated that the acute onset of hyperglycemia is the final result of a chronic preclinical phase (prediabetes). Already during prediabetes and definitely at t
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