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Titlebook: Inflammation and Metastasis; Yoshiro Maru Book 2016 Springer Japan 2016 Inflammation.Metastasis.Leukocytes.Microenvironment.Pathogen senso

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Issue of Self and Non-selfn fend off encounters with microbial toxins derived from their growth in foods. For human beings as a self, the most obvious non-self is other species, such as botany and reptiles. (king cobra) can kill humans by its neurotoxin acting on nicotinic acetylcholine receptor (nAchR) in humans [1]. In add
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Autoinflammatory Disorderss. Given that metastasis is a clinical name or name of a disease, I believe that any research irrelevant to or in disagreement to clinical information is of low value. There is no doubt that fever is one of the fundamental symptoms of ambiguously defined “inflammatory disorders” in clinical settings
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Whole-Body Matterre disease: osteosarcoma (see Table . in Chap. .). This raised the curtain on metastatic diseases. The disease spreads from the leg to the chest as a whole-body matter. Here I will discuss cancer from the standpoint of inflammation, which is basically a whole-body matter with inevitable immunologica
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Premetastatic Microenvironmenthemical, aggravates the metastatic progression of the primary tumor unless the procedures can impair the tumor cell survival at least partly. For example, skilled surgeons can remove a tumor with minimal tissue damage as represented by little blood loss during operation. Earlier removal of the prima
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Basic Researchnsformation is mediated by a RSV-dependent DNA intermediate, namely DNA provirus, stabilized in the transformed cells [3]. We should consider the fact that acute viral infection is a self-limited process for propagation of virus but persistent transformation of the infected cells needs to have some stable alterations pertinent to the cells.
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