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发表于 2025-3-21 17:36:56 | 显示全部楼层 |阅读模式
书目名称Group B Coxsackieviruses
编辑Steven Tracy,M. Steven Oberste,Kristen M. Drescher
视频video
丛书名称Current Topics in Microbiology and Immunology
图书封面Titlebook: ;
出版日期Book 2008
版次1
doihttps://doi.org/10.1007/978-3-540-75546-3
isbn_softcover978-3-642-09476-7
isbn_ebook978-3-540-75546-3Series ISSN 0070-217X Series E-ISSN 2196-9965
issn_series 0070-217X
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发表于 2025-3-21 22:59:38 | 显示全部楼层
https://doi.org/10.1007/978-3-319-14860-1The CVB have long been recognized as significant pathogens of infants and children. Although the major route for transmission of the CVB is fecal-oral, vertical transmission from mother to infant is also possible. This review will focus on the more common or clinically relevant CVB-related syndromes, their diagnosis, treatment, and prevention.
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Investigations of Anorectal Function,o explore the mechanisms by which CVB4 causes acute and chronic pancreatitis. The present review develops the idea that disease progression is a continuum, from acute inflammatory disease to chronic inflammatory disease to cancer, and that accompanying changes in gene expression are both quantitative and qualitative.
发表于 2025-3-22 21:33:40 | 显示全部楼层
R. Uday Kiran,Masashi Toyoda,Koji Zettsucontained viral myocarditis to a pathogenic autoimmune response is made within hours after induction of infection and is characterized by production of a few key cytokines, including IL-1β and TNFα. Many of the lessons learned from study of these models are applicable to human myocarditis and dilated cardiomyopathy.
发表于 2025-3-23 04:18:13 | 显示全部楼层
Comparative Genomics of the Coxsackie B Viruses and Related Enteroviruses by species (not by type), reflecting a high frequency of intertypic recombination within a species. Further genomic studies, accompanied by well-characterized clinical outcome/disease data, will facilitate fine-scale mapping of genetic determinants that contribute to virulence.
发表于 2025-3-23 09:18:28 | 显示全部楼层
CVB Translation: Lessons from the Poliovirusescation of translation factors to achieve host translation shutoff and promotion of viral translation are discussed. The translational bases for neurovirulent phenotypes and tissue specificity are also addressed.
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