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Titlebook: Diet and Atherosclerosis; Cesare Sirtori,Giorgio Ricci,Sergio Gorini Book 1975 The Editor(s) (if applicable) and The Author(s), under excl

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978-1-4615-9031-6The Editor(s) (if applicable) and The Author(s), under exclusive license to Springer Science+Busines
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Toward a Gauge Theory of Musical Forcesfic understanding of the metabolic aberrations that provoke the disease. Such a belief is perhaps difficult to sustain in view of the very slow and limited progress that has been made towards that goal over recent decades. Yet no alternatives or shortcuts present themselves. Our approaches cannot be
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Helena Àgueda Marujo,Luis Miguel Netoht weeks later it was observed that the plasma cholesterol level of the cholesterol fed birds was increased ten fold when compared to the controls on plain mash. Several birds were sacrificed and no aortic atherosclerosis was observed in any of the controls on plain mash while all those on an athero
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Claudia A. Mikkelsen,Sofía E. Aresantities of carbohydrate, mainly because they are the basis of cheap food and because they satisfy hunger, and it was the dentists that were the first to point out that dietary carbohydrates could produce pathological lesions. Then in 1961 (1) it was reported that the level of triglyceride in the fa
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T. Heindel,S. Rodt,S. Reitzenstein platelets play a predominant role (cf.55). Platelets in turn are of considerable interest as the model of a metabolically active, contractile cell, capable of reacting to a variety of external stimuli. In the course of their “activation”, they display a series of morphological and biochemical alter
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https://doi.org/10.1007/978-3-319-56378-7ning 4 different amounts of cholesterol was fed to over 60 male prisoners. The results clearly show that for each 100 mg cholesterol per 1000 calorie increment there is a linear and predictable rise in serum cholesterol. But, these plasma changes are small and do not tell us how much cholesterol is
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