Titlebook: Death Receptors in Cancer Therapy; Wafik S. El-Deiry Book 2005 Humana Press 2005 TNF.apoptosis.carcinoma.cell.cell death.gene therapy.inte

白杨鱼

condone

Ceramide, Ceramidase, and FasL Gene Therapy in Prostate Cancer,or bioactive lipids (.–.). Thus, regulation of sphingolipid metabolism appears involved in regulation of cell growth, differentiation, senescence, and programmed cell death, and possibly, as proposed herein, favoring growth of a subset of prostate cancers.

无畏

Spyridon Karageorgos,Hamid Bassiriimmune surveillance/suppression of cancer. Caspase activation is highly regulated and defects at virtually all levels of death regulation are observed in cancer. This chapter focuses on the cell biology, biochemistry, and genetics of programmed cell death.

放逐某人

泥沼

Laurence Arbibe,Philippe Sansonetting apoptosis-related proteins contribute to either development or progression of human cancers. In this chapter, we present an overview of the death receptor pathway and its dysregulation in cancers. We then review the current knowledge of death receptor mutations that have been detected in humans.

Disk199

悠然

存心

2196-9906 olecules that provide targets for developing agonists or antagonists to modulate death signaling for therapeutic purposes. The authors focus on the extrinsic system of death receptors, their regulation and function, and their abnormalities in cancer. Topics of particular interest include resistance

erythema

FLAX

https://doi.org/10.1007/978-1-4613-1137-9or bioactive lipids (.–.). Thus, regulation of sphingolipid metabolism appears involved in regulation of cell growth, differentiation, senescence, and programmed cell death, and possibly, as proposed herein, favoring growth of a subset of prostate cancers.