Titlebook: Congenital Heart Diseases: The Broken Heart; Clinical Features, H Silke Rickert-Sperling,Robert G. Kelly,David J. Dr Book 20161st edition S

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https://doi.org/10.1007/978-3-322-80473-0 of the epicardial cell lineage in coronary blood vessel morphogenesis, from the contribution of epicardially-derived mesenchymal cells to these blood vessels to its role as an instructive signaling center, attempting to relate these concepts to the origin of coronary disease.

轻浮女

Nebulous

切掉

Margit Fauser,Eveline Reisenauerulation including .-regulatory elements, chromatin structure, and micro-RNAs, which can interact with transcription factors, modulate their function, or are downstream targets. Finally, examples of disturbances of the cardiac regulatory network leading to congenital heart diseases in human are provided.

Optic-Disk

Epicardium and Coronary Arteries of the epicardial cell lineage in coronary blood vessel morphogenesis, from the contribution of epicardially-derived mesenchymal cells to these blood vessels to its role as an instructive signaling center, attempting to relate these concepts to the origin of coronary disease.

心胸开阔

Establishment of Cardiac Lateralitysignaling cascade ultimately driving left-sided expression of the Pitx2 transcription factor. We summarize recent insights gained from studies in animal models, and present a brief overview of the complex processes regulating the specification of cardiac asymmetry, and its contribution to congenital heart defects.

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Inter- and Intracellular Signaling Pathways the inter- and intracellular signaling mechanisms in the heart as they were discovered mainly in genetically modified mice. The hope is that in the future, targeted therapy of specific molecules or cascades will allow correction of defects that lead to the development of congenital heart disease or pathological hypertrophy.

多嘴

积习难改

Neural Crest discuss the contributions of the CNCCs to cardiovascular development, the signaling pathways involved in neural crest development, and some of the human congenital heart diseases attributed to aberrant CNCC development.

鉴赏家