Titlebook: Coeliac Disease; 40 years gluten-free M. L. Mearin,C. J. J. Mulder Book 1991 Springer Science+Business Media Dordrecht 1991 biopsy.endoscop

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https://doi.org/10.1007/978-3-531-91369-8also studied individual growth curves and formed three groups. Group I, 44 patients (73%), was defined by a positive change in SDS of more than 1.0. Within this group at the time of diagnosis 19 children had a SDS below −2.0 (group Ia), 22 children between −2.0–0.0 (group lb) and 3 children above 0.

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Adenovirus 12 and Coeliac Disease,be demonstrated. Despite these conflicting results on aspects of the humoral immunity to Ad12, lymphocytes of treated coeliac patients in remission, but not those of healthy subjects and patients with inflammatory bowel disease, elicited marked and dose-dependent immune responses when challenged wit

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Gliadin Peptides: Their Effect on the Gut Mucosa,hey are also able to agglutinate human K562 (S) cells, a line of chronic myeloid leukemia. The proteins and peptides that . and/or . are toxic for the coeliac small intestine are also able to damage the . developing fetal rat intestine and to agglutinate K562 (S) cells. It is likely that the unknown

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Nonhuman Primates as Animal Models for Coeliac Disease: Approach and Feasibility, it is known that these species may suffer from the same type of diseases as man. Although there exists a close phytogenetic relationship between man, great apes and monkey species the disease associated MHC immune response genes may exhibit sequence variation due to separate evolution. On first sig

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