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Titlebook: Coeliac Disease; 40 years gluten-free M. L. Mearin,C. J. J. Mulder Book 1991 Springer Science+Business Media Dordrecht 1991 biopsy.endoscop

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Intestinal Gamma/Delta Receptor Bearing T Lymphocytes are Increased in Coeliac Disease and Dermatit cells was elevated before and during gluten elimination and after the challenge test. In the epithelium, the absolute number of these cells remained constant during gluten elimination and provocation. In the specimens of DH patients a similar increase was seen; in both groups the mean number of ./.
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The Quest for Gliadin: Limits and Tolerance,nation, one must be aware of all these factors. Apart from the analytical problems in the determination of prolamins, there is the question of the amount of prolamin a patient can tolerate. We can distinguish three different actions of gliadin. First there is an allergic reaction in individuals alle
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Catch-up Growth in 60 Children with Coeliac Disease,also studied individual growth curves and formed three groups. Group I, 44 patients (73%), was defined by a positive change in SDS of more than 1.0. Within this group at the time of diagnosis 19 children had a SDS below −2.0 (group Ia), 22 children between −2.0–0.0 (group lb) and 3 children above 0.
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978-90-481-4087-9Springer Science+Business Media Dordrecht 1991
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Segregation in Language Educationt of a normal diet, wheat, is toxic to the coeliac patient. Dicke in collaboration with Van de Kamer and Weijers in Utrecht, demonstrated this with careful experiments performed on patients during different periods using different cereals. In his assay, Dr. Paveley quotes a lecture delivered by Sir
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Varied Responses to Bilingual Education11 were HLA identical, 27 haploidentical and 17 non-identical to the patient and 72 parents. For all individuals a small bowel biopsy was performed. Serological HLA typing of HLA A, B and DR antigens were done on all individuals. RFLP analysis using DRB DQA, DPA and DPB gene specific probes together
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https://doi.org/10.1007/978-3-031-32747-6deal with in vitro studies, aimed to identify the toxic proteins and peptides, to study the mechanisms of mucosal damage, and to assess the possibility of protecting the coeliac intestine from their toxic activity. The organ culture of small intestine from untreated patients has been proposed as an
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