Titlebook: Chemical Mutagens; Principles and Metho Frederick J. Serres Book 1984 Plenum Press, New York 1984 Chromosom.DNA.biology.cell lines.genotype

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Applied Economics and Policy Studies variants were proven to be due to the presence of mutant alleles by family or breeding studies, population frequencies, and electrophoretic patterns in heterozygotes consistent with the subunit structures of the enzymes (see Harris. for review).

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The Use of Multiply Marked , K12 Strains in the Host-Mediated Assay,us mammalian organ fractions, have yielded results on a variety of environmental compounds that show definite mutagenic activity in this assay system and are therefore to be considered as potentially mutagenic and carcinogenic in animals (see also Refs. 6 and 7).

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The Grasshopper Neuroblast Short-Term Assay for Evaluating the Effects of Environmental Chemicals othe current tests have been in use since the beginning of the present era of active research on the identification of environmental mutagens and carcinogens, which began to attain international momentum in the late 1960s.. Why, then, did we recently develop another assay? First, the neuroblast (Nb)

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Comparison of the Mutagenic Responses of Lung-Derived and Skin-Derived Human Diploid Fibroblast Popns was established simultaneously from the same individual using carefully controlled, identical conditions. These paired populations provide a unique opportunity to study differences between normal diploid fibroblast populations from skin and lung without the confusion of genetic differences betwee

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