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Titlebook: Cerebral Ischemia and Calcium; Alexander Hartmann,Wolfgang Kuschinsky Conference proceedings 1989 Springer-Verlag Berlin Heidelberg 1989 P

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https://doi.org/10.1007/978-3-662-30501-0robic glycolysis, retention of metabolic end-products (such as lactic acid), and depressed function [5]. The latter, in the case of the brain, is usually reflected in disappearance of delta waves in the electroencephalogram [1].
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Immunocytochemical Studies on Some Brain Regions in a Rat Model of Incomplete Cerebral Ischemia. RelSeveral pathogenetic mechanisms have been proposed to explain the peculiar histological features of brain ischemia, i.e., the selective neuronal death and the so-called maturation phenomenon [17] — the extension of the lesions during the reperfusion.
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https://doi.org/10.1007/978-3-642-85863-5PET; aneurysm; biochemistry; brain; brain injury; cerebral ischemia; cortex; depression; electroencephalogra
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978-3-642-85865-9Springer-Verlag Berlin Heidelberg 1989
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