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Titlebook: Cerebral Ischemia and Calcium; Alexander Hartmann,Wolfgang Kuschinsky Conference proceedings 1989 Springer-Verlag Berlin Heidelberg 1989 P

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书目名称Cerebral Ischemia and Calcium
编辑Alexander Hartmann,Wolfgang Kuschinsky
视频videohttp://file.papertrans.cn/224/223306/223306.mp4
图书封面Titlebook: Cerebral Ischemia and Calcium;  Alexander Hartmann,Wolfgang Kuschinsky Conference proceedings 1989 Springer-Verlag Berlin Heidelberg 1989 P
描述Although many books deal with isolated problems of calcium disturbance in relation to cardiac and cerebral function, this is the first to focus specifically on calcium metabolism and cerebral ischemia. Internationally acknowledged experts present recent data and new concepts in an interdisciplinary approach to the subject. They examine basic information on the pathophysiology of cellular events, the damage caused by excitotoxic substances, and the effects of calcium antagonistic drugs as measured in experimental animals. These contributions may help lead to possible therapeutic interference for the prevention of calcium imbalance.
出版日期Conference proceedings 1989
关键词PET; aneurysm; biochemistry; brain; brain injury; cerebral ischemia; cortex; depression; electroencephalogra
版次1
doihttps://doi.org/10.1007/978-3-642-85863-5
isbn_softcover978-3-642-85865-9
isbn_ebook978-3-642-85863-5
copyrightSpringer-Verlag Berlin Heidelberg 1989
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Pharmacological Activities of Calcium Antagonists: Therapeutic Applications and Adverse Reactionsnce as tools in fundamental research is generally recognized as well. More recently, certain types of calcium antagonists have been studied as potentially useful drugs in the treatment of neurological disorders, such as migraine, cerebral ischemia and hypoxia, certain forms of vertigo, epilepsy, and subarachnoid hemorrhage.
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Organisch-Chemische Arzneimittelanalyseognition of a class of drugs does not imply that drugs belonging to a given class have an identical pharmacological profile. Differences may be the basis for recognition of subclasses. The major common properties as well as the subtle differences need to be characterized using well established quant
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https://doi.org/10.1007/978-3-662-30501-0 evoked potential amplitude and extracellular potassium homeostasis to reductions in local blood flow. Where flow was reduced, but remained above a critical threshold for evoked potential amplitude suppression, such suppression could nevertheless be induced by haemorrhagic hypotension, and was follo
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