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Titlebook: Cerebral Ischemia and Calcium; Alexander Hartmann,Wolfgang Kuschinsky Conference proceedings 1989 Springer-Verlag Berlin Heidelberg 1989 P

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H. Pierer,D. Kronberger-Schöneckerty of the ischemic frontal cortex has been reported as early as 30 min after permanent middle cerebral artery (MCA) occlusion in the rat [22]. Fluid accumulation is believed to occur in response to an increase of sodium into the lesion site [27] or reduction in sodium clearance from the tissue [12].
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Zur plastischen Korrektur der Syndaktyliehis occlusion [1, 3, 6, 8, 10, 11, 13]. Following the occlusion in normotensive rats, the anastomosing collaterals enlarge to provide blood supply to the territory of the occluded MCA [9, 12, 15]. Within a month after the occlusion, blood flow to the territory of the occluded MCA returns to normal levels in the normotensive rat [15].
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Blood-Brain Barrier Permeability to Micromolecules After Focal Cerebral Ischemiaty of the ischemic frontal cortex has been reported as early as 30 min after permanent middle cerebral artery (MCA) occlusion in the rat [22]. Fluid accumulation is believed to occur in response to an increase of sodium into the lesion site [27] or reduction in sodium clearance from the tissue [12].
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The Classification of Calcium Antagonistsognition of a class of drugs does not imply that drugs belonging to a given class have an identical pharmacological profile. Differences may be the basis for recognition of subclasses. The major common properties as well as the subtle differences need to be characterized using well established quant
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