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Titlebook: Cardiovascular Biology of Purines; Geoffrey Burnstock,James G. Dobson,Joel Linden Book 1998 Springer Science+Business Media New York 1998

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Neuromodulation in the Cardiovascular System,bit transmitter release via prejunctional A. receptors thereby diminishing α. adrenoceptor-mediated vasoconstriction. Presumably, not only adenosine but also ATP inhibits transmitter release through activation of presynaptic A. receptors which might explain various observations previously ascribed t
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Adenosine Receptors, Blood Vessel and Inflammation,-10. These adenosine-mediated changes in PMN and moncyte/macrophage function have the potential to alter the course of an inflammatory response in cardiovascular diseases such as ischemia/reperfusion injury, atherosclerotic plaque development and inflammatory aneurysm formation.
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Purinoreceptors and T Cell Function: Regulation of Lymphocyte Activation by Extracellular ATP and Ac T cells (CTL) and T-helper cells. Evidence to support this model is suggested by studies of the contribution of purinergic receptor expression to the . functional responses of T cells. It has been shown that some purinergic receptors in T cells are expressed in a differentiation-dependent manner (
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Cardiovascular Biology of Purines978-1-4615-5603-9Series ISSN 0166-9842
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0166-9842 Overview: 978-1-4613-7563-0978-1-4615-5603-9Series ISSN 0166-9842
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Nuclear Energy and International Cooperation 1929]. These potent properties and its short half life in vivo have allowed adenosine to establish its place as a unique and invaluable tool for both elucidating the mechanism of cardiac arrhythmias and for treating supraventricular tachycardia.
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978-1-4613-7563-0Springer Science+Business Media New York 1998
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Adenosine and Cardiac Arrhythmias, 1929]. These potent properties and its short half life in vivo have allowed adenosine to establish its place as a unique and invaluable tool for both elucidating the mechanism of cardiac arrhythmias and for treating supraventricular tachycardia.
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