Titlebook: Cancer Therapy; Differentiation, Imm Natale D’Alessandro,Enrico Mihich,Thomas R. Tritto Conference proceedings 1993 Springer-Verlag Berlin

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Differential Effects of Low Doses Of Structurally Different Anthracyclines on Immunoglobulin ProducDOX induces the blockade of cell proliferation through an accumulation of the cells in the G2 + M phase of the cell cycle as well as a strong increase of the Immunoglobulin (Ig) production (Teillaud et al., 1989), suggesting a terminal differentiation in “plasma-like” cells.

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Chemical Xenogenization of Experimental Tumors by Antineoplastic Drugs,posure to the antitumor agent dacarbazine, would become increasingly immunogenic, eventually acquiring a degree of foreignness capable of resulting in tumor cell rejection by the histocompatible host.

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Colon Cancer Cell Differentiation as Related to Methotrexate and 5-Fluorouracil Resistance,herapy. Although it has been assumed that drug resistance could be associated with some particular cell populations, these have not been characterized yet. It is only in recent years that progress in the field of intestinal cell biology, based on the development of cultured cell lines and availabili

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The Control of Cell Multiplication and Differentiation in Human Myelomonocytic Cells,her susceptible or resistant to such a differentiation induced by phorbol 12-myristate 13- acetate (PMA). Unlike the cells from susceptible cell lines, the PMA-resistant variants did not express the genes that code for the protein kinase C (PKC) β isozyme and a “δ-like” PKC. The resistant cells also

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Signal Transduction Mechanisms as a Target for Cancer Chemotherapy,devoted to understanding its mechanism of action. As summarized in Table 1, several proposals have been offered to explain the anticancer activity of adriamycin and other members of the anthracycline family.

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Therapeutic Approaches for Colon Cancer Based on Transcriptional Regulation of Specific Growth Fact a consequence, this tumor has been the subject of extensive investigations seeking alternative or perhaps supplemental therapies based on biochemical, biological or immunological properties of colon cancer cells which are not shared by normal cells.

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