Titlebook: Brain Damage and Repair; From Molecular Resea T. Herdegen,J. Delgado-García Book 2004 Springer Science+Business Media B.V. 2004

Occipital-Lobe

Minikin

Neural Plasticity and Regeneration: Myths and Expectations, which are sometimes confused with neural plastic processes. As a practical support to the concepts proposed here, some illustrative examples, collected from animal experimentation carried out in our laboratory, will be described briefly.

INCH

imer, M. Parkinson, ALS, stroke or trauma, take this knowled.Brain Damage and Repair. aims to attract the interest of neuroscientists and clinical neurologists by building a novel bridge from molecular research to clinical therapy. This novel approach reveals the functional features of neurons and g

Override

Conclusions, Suggestions and Prospects,herapeutic targets for demyelinating diseases. We also summarize some relevant advances in the etiology of multiple sclerosis, the most frequent disease involving oligodendrocyte and myelin loss, and new concepts as to how white-matter damage during ischemia contributes to the clinical outcome of stroke in humans.

皮萨

ESPY

Oligodendrocytes in Health and Diseaseherapeutic targets for demyelinating diseases. We also summarize some relevant advances in the etiology of multiple sclerosis, the most frequent disease involving oligodendrocyte and myelin loss, and new concepts as to how white-matter damage during ischemia contributes to the clinical outcome of stroke in humans.

适宜

Neuronal Protection Against Oxidative Damage species, oxidative damage may occur. Such damage may be counteracted by superoxide dismutases, glutathione peroxidase, and metallothioneins. Antioxidant protectors can be generated by ascorbic acid and a-tocopherol. Oxidative mechanisms in neurodegenerative diseases are exemplified by Alzheimer’s disesase.

伪证

Book 2004 ALS, stroke or trauma, take this knowledge, apply it at the clinical level to provide a basis for novel therapeutic strategies. .Brain Damage and Repair. is completed with chapters on imaging techniques, viral gene transfer or legal international handling of stem cell transfer and patents of DNA/proteins.

Gorilla

https://doi.org/10.1007/978-88-470-1060-4cium blockers, a variety of growth factors, immune suppressants, antiinflammatory agents, as well as free radical scavengers. The recent identification of a gene, which “protects” axons from undergoing degeneration, provides a basis for a genetic approach to the treatment of neurological disorders caused by axonal disease or trauma.

CRANK

https://doi.org/10.1007/978-981-97-4220-2ading edge; and through intracellular kinases, which have the potential to alter gene expression changes in the developing neuron. In addition, we also summarize the increasing body of knowledge on their roles in the inhibition of axon regeneration after adult lesions in the central and peripheral nervous system.