Titlebook: Apoptosis in Immunology; Guido Kroemer,Carlos Martinez-A. Book 1995 The Editor(s) (if applicable) and The Author(s), under exclusive licen

d-limonene

Cenni sulla dinamica della turbolenza,t al. 1972; . and . 1991; . and . 1993a; . and . 1992), but briefly, apoptosis involves separation of the cell from its neighbors, condensing of the cytoplasm, condensing of the chromatin, which moves to the margins of the nuclear envelope, convolution of the plasma membrane, and, finally, blebbing

exorbitant

,L’approssimazione di strato limite, size (. and . 1974). Glucocorticoid-induced apoptosis is dependent on the binding of glucocorticoids to glucocorticoid hormone receptors (GRs), which is also required in the general effects of steroids (. et al. 1984). The steroid receptors such as GR, mineralcorticoid receptors, progesterone recep

miracle

Anthem

紧张过度

GILD

Roberto Grassi,Giovanni Pinto,Alfredo Siani hormones), and the particular subpopulation to which the responding cells belong and their differentiation stage and (pre-) activation state. In this sense, T cells function as semiotic entities; they integrate signals from a complex universe as a function of their previous experiences to act in a

有发明天才

Book 1995the signal transduction pro­ cesses involved therein and describe the role of oncogenes, "death genes" and effector molecules in the apoptotic cas­ cade. The remaining chapters deal with the pathophysiologi­ cal aspects of lymphocyte apoptosis, namely, as a host contribution to HIV-induced lymphopen

Creditee

peak-flow

,L’approssimazione di strato limite,ignals including: (1) γ-irradiation (. and . 1987); (2) glucocorticoids (. 1980; . and . 1989); (3) cell surface signaling via the T cell receptor (TCF)/CD3 Complex (. et al. 1989; . et al. 1989; . et al. 1989; . et al. 1991; . et al. 1991, 1992), via the CD2 antigen (. et al. 1991; . et al. 1993a),

pulse-pressure

,L’approssimazione di strato limite,act to self-antigens. The development of B and T lymphocytes is therefore controlled at multiple levels in order to ensure that individuals will be able to respond to a large array of foreign antigens while being tolerant to self. Under some circumstances, the encounter between a foreign antigen and