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Titlebook: Spectral Readings; Towards a Gothic Geo Glennis Byron (Senior Lecturer in English Studies) Book 1999 Palgrave Macmillan, a division of Macm

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楼主: CHARY
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Alexandra Warwickressor Snail. NO-induced inhibition of NF-κB and Snail and induction of RKIP established the NO-induced dysregulation of the NF-κB/Snail/RKIP circuitry in the regulation of metastasis. These findings suggest the therapeutic role of NO donors in the inhibition of metastasis.
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Robert Mighallduction of cytotoxicity by NO in vivo can boost T-cell responses by partially degrading tumor cells and thus facilitate antigen presentation of APC. Furthermore NO is essential in the early stages of T-cell activation. However prolonged secretion of NO can also induce tolerance and/or immunosuppress
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Helen F. Thompsoned. Moreover, iNOS and COX-2 have been found to be co-expressed within the same tumor cells and involved in the regulation of tumor growth. In conclusion, iNOS and COX-2 products may represent a common final pathway controlling different tumorigenic mechanism.
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Eric Savoy to participate in a plethora of relevant molecular and cellular reactions even in eATP-mediated responses. In this chapter, we shortly reviewed the participation of eATP and NO as players of an orchestrated complex net of events occurring downstream wounding in plants. A brief discussion on the int
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Barnard Turneress elevates synthesis of NO which in association with these molecules stimulate the DNA-binding activity of HSFs as well as the accumulation of HSPs leading to resistance to heat stress. Moreover, exogenous application of NO has also been shown to have an ameliorative effect on heat stress, which h
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