Titlebook: Oncogenes and Tumor Suppressor Genes in Human Malignancies; Christopher C. Benz,Edison T. Liu Book 1993 Kluwer Academic Publishers 1993 DN

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Growth regulation of human neuroblastoma,ignancies among infants [for review see .,.]. A significant fraction of cases are identified neonatally, indicating that the tumor can arise during fetal life and may represent a disorder of normal development [.]. These tumors arise in sympathetic neuroblasts that originate in the neural crest and

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Malignant transformation by , and ,,an feline sarcoma virus II, and, through the Philadelphia chromosome, are linked with the myeloproliferative syndrome chronic myelogenous leukemia and with acute lymphoid and myeloid leukemia in humans. This review will outline the current state of knowledge concerning this important oncogene family

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Transforming growth factor-alpha and its role in neoplastic progression,formed cell lines showed reduced surface binding of radiolabeled EGF. This led to an examination of media conditioned by these cells for factors that would bind the EGF receptor, thereby making it unavailable to the exogenously added ligand. Such a factor was identified in media conditioned by felin

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,Growth regulation by transforming growth factor-β,ies. The first member of this gene family was identified nearly a decade ago as one of two essential factors, called TGF-α and TGF-ß present in the conditioned medium of a murine sarcoma virus-transformed cell line, which together stimulated the anchorage-independent growth of non-transformed fibrob

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Signal transduction by receptor tyrosine kinases,pagate and amplify signals received from the environment to specific targets within the cell. The culmination of this pathway is DNA synthesis and cell division. Since growth is not a common event in organs of mature multicellular organisms, these pathways must be precisely regulated. The signalling

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