Titlebook: New Trends in Cancer for the 21st Century; Proceedings of the I Antonio Llombart-Bosch,Vicente Felipo Conference proceedings 2003 The Edito

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Structural Basis of Tumoral Angiogenesis,ply of these needs within 200 to 250µm. Multicellular organisms that grow beyond this size require the recruitment of new blood vessels, although some normal tissues are devoid of specific vascularization (cartilage, cornea, epidermis), obtaining their oxygen and metabolic supply through perfusion

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MUMP

New Trends in Cancer for the 21st Century978-1-4615-0081-0Series ISSN 0065-2598 Series E-ISSN 2214-8019

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Driving the Cell Cycle to Cancer,east four of these so-called cyclin-dependent kinases, namely Cdk4, Cdk6, Cdk2 and Cdkl, have specific roles at particular stages of the cell cycle, including passage through the various cell cycle transitions and the response to specific checkpoints. Not surprisingly, most human tumors carry mutati

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Proliferation: The Cell Cycle,ons, whereas the majority of immortal cells used in laboratory cultures have acquired mutations during the immortalization process and are susceptible to being transformed. Our main interest is the study of the molecular mechanisms which signal anti-tumor responses as well as the impact these anti-t

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Ewing Tumor Biology: Perspectives for Innovative Treatment Approaches,ene rearrangement between the.gene and an.transcription factor gene. EFT can be cured with conventional multi modal treatment, however, about 40% of patients still succumb to the disease. Relapses can be observed more than 5 years after the end of primary treatment suggesting persistence of minimal

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Cancer Epigenetics: DNA Methylation and Chromatin Alterations in Human Cancer,ethylation of CpG islands of tumor suppressor genes associated with their transcriptional silencing The target genes are distributed in all cellular pathways (apoptosis, DNA repair, cell cycle, cell adherence, etc) They are “classical” tumor suppressor genes with associated familial cancers (BRCA1,

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