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Titlebook: Neuroproteomics; Ka Wan Li Book 2019Latest edition Springer Science+Business Media, LLC, part of Springer Nature 2019 rodent brain regions

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TMT-MS3-Enabled Proteomic Quantification of Human IPSC-Derived Neurons, Alzheimer’s, and Parkinson’s disease can be reproducibly modeled. Proteomic analysis of these model systems has the potential to identify disease signatures, characterize treatment effects, and drive biomarker identification. Here we describe the implementation of a TMT-MS.-based proteomic workflow
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Quantitative Analysis of Mass Spectrometry-Based Proteomics Data,tein report and statistical analysis. We use a publicly available dataset as a working example that covers two prominent strategies for mass spectrometry-based proteomics, the extensively used data-dependent acquisition (DDA) and the emerging data-independent acquisition (DIA) technology. We use Max
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Neuropeptidomics of the Mammalian Brain,mics evolved from the quest to globally characterize these important cell–cell signaling peptides. The goal of a neuropeptidomics experiment is to characterize the peptides present in an intact brain, brain region, or even an individual neuron. A neuropeptidomics measurement needs to contend with th
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A Combined Cellomics and Proteomics Approach to Uncover Neuronal Pathways to Psychiatric Disorder,s complexity requires research approaches to reliably dissect the cell biology of the risk genes involved. Here, we describe a combined cellomics–proteomics approach that allows (a) medium-throughput functional screening and unbiased selection of important risk genes, and (b) discovery of common fun
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