Titlebook: Neuroprotective Signal Transduction; Mark P. Mattson Book 1998 Springer Science+Business Media New York 1998 Deprivation.Nervous System.Pa

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,Signaling by β-Amyloid Precursor Protein,brane glycoprotein having a single transmembrane domain, a large extracelluar N-terminus, and a short cytoplasmic C-terminus (Kang et al., 1987). This protein is very important as a source of amyloid β peptide (Aβ), a 40–42 amino acid peptide that forms senile plaques in Alzheimer’s disease (AD) pat

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Clinical Potential of Agents That Affect Thrombin Signaling in Degenerative and Traumatic Neurologigens for other enzymes involved in coagulation. The most well known, in this context, is Factor XIII activation to XIIIa. Thrombin catalyzes this reaction, activating a powerful Ca.-dependent enzyme for crosslinking fibrin via the γ-carboxamide group of glutamine residues and the ε-amino group of ly

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Therapeutic Potential of Leukocyte Antiadhesion Strategies in CNS Ischemia,ting injury. Recent evidence suggests that leukocytes may also be directly involved in the pathogenesis and extension of CNS ischemia/reperfusion (I/R) injury (Engler et al., 1986; Schmid-Schonbein and Engler, 1986). Two proposed mechanisms of leukocyte involvement in IIR are: direct microvascular o

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Neuroprotective Strategies Based on Targeting of Postreceptor Signaling Events, may be involved in various neurodegenerative conditions. This last chapter is a signal transduction hodgepodge, reviewing some of the same neuroprotective signaling pathways discussed in other chapters, as well as presenting novel neuroprotective signaling mechanisms not considered elsewhere. It em

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ment involves deliberate and pre-planned intervention by outside agencies to achieve economic growth, immanent development occurs as an outcome of the penetration of capitalism. Intentional development represents a form of development which is commonly recognized, such as state-sponsored development

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