Titlebook: Neuroendocrinology of Gastrointestinal Ulceration; Sandor Szabo,Yvette Taché,Gary B. Glavin Book 1995 Springer Science+Business Media New

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The Development of Acute Gastric Mucosal Lesions Following Burn Stressgastric mucosal blood flow. Therefore, blood flow is an extremely important defensive factor for the gastric mucosa. The present study was designed to elucidate the role of mucosal blood flow in the development of acute gastric mucosal lesions (AGML) after burn stress. Gastric mucosal blood flow and

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Experimentally-Induced Duodenal Ulcers in Rats are Associated with a Reduction of Gastric and Duoden (li) is widely distributed in the rat gastrointestinal tract. and high concentrations of CGRP-li have been found in the rat stomach and duodenum.. The absence of CGRP-li in endocrine cells. at these levels suggests that it is more important as a neuropeptide than as a circulating hormone. Exogenous

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The Effect of bFGF and PDGF on Acute Gastric Mucosal Lesions, Chronic Gastritis and Chronic Duodenalstric acid or inhibiting acid secretion. In these cases the erosions and ulcers were essentially left alone to heal themselves in the presence of diminished aggressive factors, but without any attempt of stimulating the healing process.

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Animal Models and Pathogenesis of Inflammatory Bowel Diseaseance, it remains unclear whether those alterations in immunologic function (such as autosensitization to epithelial cell components). or in the activity or structure of the enteric nervous system (degeneration of the autonomic nerves). which are characteristic of IBD represent initiating factors or

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Leukotrienes in Human and Experimental Ulcerative Colitisated with the use of non-steroidal anti-inflammatory drugs, ulceration of the more distal regions of the gastrointestinal tract are also an important clinical problem. Inflammatory bowel disease (IBD), which is an umbrella term for Crohn’s disease and ulcerative colitis, is characterized by chronic

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Neuroendocrine Control of Gastric Acid Secretionre to a great extent regulated by neurogenic mechanisms, considerable attention has been focused on the possible role of the hypothalamus and other regions of the brain in stress. Actually there was little information regarding the relationship of central neurotransmitters to gastric functions, part

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Stress, Corticotrophin-Releasing Factor (CRF) and Gastric Functionme the number of methods for producing GE’s in the rat has grown exponentially.. Initially, the burden of the pathogenesis of GE was placed on the corticosteroids and on increases in gastric acid secretion (GAS). Yet, Cannon in 1929 first showed that GAS was reduced in the threatened cat. Since that