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Titlebook: Neuroendocrinology of Gastrointestinal Ulceration; Sandor Szabo,Yvette Taché,Gary B. Glavin Book 1995 Springer Science+Business Media New

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书目名称Neuroendocrinology of Gastrointestinal Ulceration
编辑Sandor Szabo,Yvette Taché,Gary B. Glavin
视频video
丛书名称Hans Selye Symposia on Neuroendocrinology and Stress
图书封面Titlebook: Neuroendocrinology of Gastrointestinal Ulceration;  Sandor Szabo,Yvette Taché,Gary B. Glavin Book 1995 Springer Science+Business Media New
出版日期Book 1995
关键词Flow; neuroendocrinology; neurons; neuropeptides; physiology
版次1
doihttps://doi.org/10.1007/978-1-4615-1867-9
isbn_softcover978-1-4613-5759-9
isbn_ebook978-1-4615-1867-9
copyrightSpringer Science+Business Media New York 1995
The information of publication is updating

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Overview: 978-1-4613-5759-9978-1-4615-1867-9
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Corticotropin Releasing Factor and Gastric ErosionsSeveral neuropeptides have been shown to either promote or inhibit the development of gastric erosions in rats; recent reviews have been presented by Taché. and Hernandez..
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Role of the Vagal Nerve in the Development of Gastric Mucosal Injury and its Prevention by Atropine,Peptic ulceration including acute erosions is a multifactorial disease involving well-known factors such as trauma, stress, sepsis, hemorrhagic shock, burns, pulmonary and liver diseases. and drugs such as reserpine., epinephrine., nonsteroidal antiinflammatory drugs., steroids and other chemicals..
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The Effect of bFGF and PDGF on Acute Gastric Mucosal Lesions, Chronic Gastritis and Chronic Duodenalstric acid or inhibiting acid secretion. In these cases the erosions and ulcers were essentially left alone to heal themselves in the presence of diminished aggressive factors, but without any attempt of stimulating the healing process.
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Hans Selye Symposia on Neuroendocrinology and Stresshttp://image.papertrans.cn/n/image/664030.jpg
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Role of Sensory Neurons in the Control of Gastric Mucosal Blood Flow and Protection and of both endogenous and exogenous origin. Most tissues would rapidly disintegrate if exposed to the concentrations of hydrochloric acid that bathe but do not harm the gastric surface epithelium. This is because a multitude of mechanisms constituting the gastric mucosal barrier prevents hydrogen
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