Titlebook: NF-kB in Health and Disease; Michael Karin Book 2011 The Editor(s) (if applicable) and The Author(s), under exclusive license to Springer-

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Anurupa Dev,Shankar Iyer,Bahram Razani,Genhong Chengcoupling- are fundamental for the prospects of sustainability..This book provides a valuable resource for economists, energy and environmental analysts, natural resource managers and policy makers. It is also i978-3-319-20731-5978-3-319-20732-2Series ISSN 2191-5504 Series E-ISSN 2191-5512

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,NF-κB/Rel Proteins and the Humoral Immune Responses of ,,anisms of microbial recognition, signal transduction and NF-κB regulation, in both the Toll and the IMD pathways. Similarities and differences relative to their mammalian counterparts are discussed, and recent advances in our understanding of the intricate regulatory networks in these NF-κB signalin

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,Macrophages and NF-κB in Cancer,on in TAM has, therefore, been suggested to represent a molecular link between inflammation and cancer. However, TAM frequently display an anti-inflammatory phenotype linked with immune-suppression that is not easily reconciled with a pro-inflammatory function for NF-κB in TAM. Here, I review the fo

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,NF-κB as a Target for Oncogenic Viruses,ar virus. These viruses encode specific oncoproteins that target different signaling components of the NF-κB pathway, leading to persistent activation of NF-κB. This chapter will discuss the molecular mechanisms by which NF-κB is activated by the viral oncoproteins.

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,Understanding the Logic of IκB:NF-κB Regulation in Structural Terms,rom a small cadre of five gene products that assemble with one another in various combinations to form active homo- and heterodimers. The ability of NF-κB to alter target gene expression is regulated at many levels. Chief among these regulatory mechanisms is the noncovalent association in the cell c

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Specialized Chromatin Patterns in the Control of Inflammatory Gene Expression, expression programs display two important features: first, alternative stimuli affect both a large set of common genes and a variable number of stimulus-specific genes. Second, different genes are activated with different kinetics over several hours after the initial stimulus. Mechanistically, thes