Titlebook: Male-Mediated Developmental Toxicity; Andrew F. Olshan,Donald R. Mattison Book 1994 Plenum Press, New York 1994 behavior.cancer.developmen

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Male Mice Receiving Very Low Doses of Ionizing Radiation Transmit an Embryonic Cell Proliferation Di of this ‘chimera assay’ is its exquisite sensitivity for detecting male germ cell exposure to ionizing radiation. Its published detection limit for male germ cell exposure is presently 0.01 Gy exposure of low LET radiation (e.g., x-radiation and gamma radiation). An important feature of the chimera

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The Male-Mediated Developmental Toxicity of Cyclophosphamided primarily on the consequences of maternal exposure to teratogens; much less attention has been given to the possible adverse effects on the progeny of paternal exposure to drugs or environmental chemicals. However, a wide range of agents, including environmental pollutants (e.g. lead) and therapeu

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Male-Mediated Teratogenesis: Ionizing Radiation/Ethylnitrosourea Studiesations result in these defects. Animal experiments anticipated that parental exposure to radiation and chemicals would induce varieties of defects in the offspring, including embryonic deaths, tumors and malformations (Nomura, 1975; 1978; 1982; 1986; Tomatis, 1981). However, these findings have not

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Preconception Exposure of Males and Neoplasia in their Progeny: Effects of Metals and Consideration rethane, ethylnitrosourea, 4-nitroquinoline N-oxide, diethylstilbestrol, or cyclophosphamide), have resulted in significant increases in the incidences of tumors in their progeny and sometimes later generations (review in Tomatis et al, 1992; see also Francis et al, 1990; Loktionov et al.; 1992, Tur

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Male-Mediated Developmental Toxicity: Paternal Exposures and Childhood Canceress of carcinogenesis as it applies to both adults and children. It can also be a frustrating and unrewarding pursuit. The question of the effect of paternal exposure on risk of cancer in an offspring illustrates both the fascination of this field, and its limitations.