Titlebook: Inhibitory Regulation of Excitatory Neurotransmission; Mark G. Darlison Book 2008 Springer-Verlag Berlin Heidelberg 2008 G-Protein Coupled

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Regulation of Excitability by Extrasynaptic GABAA Receptors, ambient, extracellularGABA as a more persistent form of signalling (often termed tonic inhibition). Over the last decadetonic GABA. receptor-mediated inhibition and the properties of GABA.receptors mediating this signalling have received increasing attention. Tonic inhibition is present throughoutt

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The Role of GABAB Receptors in the Regulation of Excitatory Neurotransmission,they are made up of a dimerof 7-transmembrane spanning subunits. The receptors are widely distributed throughout the central nervoussystem where they act post-synaptically to cause a long-lasting hyperpolarisation through the activationof a potassium conductance. They are also present pre-synaptical

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GABAergic Control of CA3-driven Network Events in the Developing Hippocampus,s known as “Giant Depolarizing Potentials”(GDPs) are seen . at a stage when GABAergic transmissionis depolarizing. GDPs are triggered by the CA3 region and they are seen as brief recurrent events in field-potentialrecordings, paralleled by depolarization and spiking of pyramidal neurons. In the ligh

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Regulation of Excitation by Glycine Receptors,rogeneous, and undergo developmental changes. Activationof GlyRs of immature neurons induces outflow of Cl.,membrane depolarization, neuronal excitation, calcium influx, and transmitter release, in contrast to theinhibitory effects these receptors have in mature neurons. Thus, GlyRs are important fo

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Regulation of Excitability by Potassium Channels,diversity reflects the need to regulate and fine-tuneneuronal excitability at various levels of complexity in space and time. In this context, Kv channels operatingin the subthreshold range of action- potential firing are of particular interest. It is likely that theseKv channels play a prominent ro

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