Titlebook: Inflammatory Mechanisms in Mediating Hearing Loss; Vickram Ramkumar,Leonard P. Rybak Book 2018 Springer International Publishing AG, part

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Peter S. Steygerw to choose or adapt a control architecture and design or analyse it on a systematic basis... ..Features:..associated website contains many ready-made examples and case studies using Matlab®/Simulink® to which 978-1-85233-387-4978-1-4471-0047-8Series ISSN 1439-2232 Series E-ISSN 2510-3814

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Book 2018ut none of these have been approved by the US Food and Drug Administration for the treatmentof hearing loss.. More recently, it was shown that noise-induced hearing loss was associated with recruitment of inflammatory cells and mediators in the cochlea. This finding would suggest that noise could pr

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The Cochlea,at permit this organ to function properly. This chapter describes some of the features of the cochlea including the cells that line the fluid filled spaces of the cochlear duct and the chemical composition of the fluids that allow the tissues to produce resting and acting potentials that assist in t

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Oxidative Stress and Hearing Loss,ative stress as well as an overview of cochlear oxidative stress pathways activated by risk factors of auditory dysfunction. It also discusses the susceptibility of the inner ear to oxidative damage, the intracellular redox sensitive mechanisms that facilitate cytotoxicity, and the cochlear targets

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Corticotropin Releasing Factor Signaling in the Mammalian Cochlea: An Integrative Niche for Cochleanto neural responses sent onward to the brain. Many of these “support” cells exist in niches that position them to monitor the state of the cochlea, and some are situated to signal such information to systems outside the cochlea. Others occupy positions such that they can invoke cellular responses l

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Cochlear Inflammation Associated with Noise-Exposure,ra, intensities and durations, we know relatively little about the inflammatory response to these noises. Some cochlear cells up-regulate their expression of inflammatory mediators in response to noise and presumably thereby, recruit circulating macrophages into the cochlea or activate resident cell