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Titlebook: Inflammation; John R. Vane,Sergio H. Ferreira Book 1978 Springer-Verlag Berlin Heidelberg 1978 Chemokine.Histamin.Hypothalamus.Insulin.Nic

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发表于 2025-3-21 18:11:20 | 显示全部楼层 |阅读模式
书目名称Inflammation
编辑John R. Vane,Sergio H. Ferreira
视频video
丛书名称Handbook of Experimental Pharmacology
图书封面Titlebook: Inflammation;  John R. Vane,Sergio H. Ferreira Book 1978 Springer-Verlag Berlin Heidelberg 1978 Chemokine.Histamin.Hypothalamus.Insulin.Nic
描述Throughout the centuries, inflammation has been considered as a disease in itself. This misconception arose from the inability to distinguish between inflammatory changes and the insults which induce them. The understanding of the distinction between the genesis of inflammation and the tissue reactions that follow is attributed to JOHN HUNTER, who, at the end of the 18th century, substantially contributed to the analysis of inflammation in objective terms. Today, however, we are still trying to find explanations for Celsus‘ Signs in terms of structural and functional changes occurring in the inflamed tissue. There are drugs which modulate these signs but, without a detailed knowledge of the basic physiopathological events, it is impossible to understand their mechanism of action. Notwithstanding, the effects of anti­ inflammatory drugs provided new knowledge of the relevance of the signs and symptoms to the sequence of biochemical and morphological changes occurring in inflammation. When we accepted the invitation to edit a Handbook on Inflammation and Anti­ Inflammatory Drugs, we were aware of the magnitude of the task. We knew the impossibility of covering the whole field in deta
出版日期Book 1978
关键词Chemokine; Histamin; Hypothalamus; Insulin; Nicotin; Prostaglandin; antibody; antigen; asthma; biochemistry; b
版次1
doihttps://doi.org/10.1007/978-3-642-66888-3
isbn_softcover978-3-642-66890-6
isbn_ebook978-3-642-66888-3Series ISSN 0171-2004 Series E-ISSN 1865-0325
issn_series 0171-2004
copyrightSpringer-Verlag Berlin Heidelberg 1978
The information of publication is updating

书目名称Inflammation影响因子(影响力)




书目名称Inflammation影响因子(影响力)学科排名




书目名称Inflammation网络公开度




书目名称Inflammation网络公开度学科排名




书目名称Inflammation被引频次




书目名称Inflammation被引频次学科排名




书目名称Inflammation年度引用




书目名称Inflammation年度引用学科排名




书目名称Inflammation读者反馈




书目名称Inflammation读者反馈学科排名




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Complemente. Detailed studies of the human complement sequence have facilitated the demonstration of acquired abnormalities in association with disease, while an understanding of the system in certain animals has provided models for experimental studies of the role of complement in inflammation.
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Bradykinin-Systemradykinin (Bk), in such reactions will be discussed. The main scope is to evaluate to what extent kinins are contributing to the inflammatory response, as well as to assess the significance of their participation in such a complex, multimediated process.
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The Sequence of Early Eventsd by all other techniques—histological, ultrastructural, biochemical or pharmacological—must be compared in order to determine its relevance to the morphology and mechanisms of the tissue response to injury.
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Immunological and Para-Immunological Aspects of Inflammationconcerned with a review of the immunological causes of inflammation and, in addition, with those phenomena which may lack the specificity normally associated with the “specific adaptive immune response” but which use some of the pathways normally associated with immunological reactions, such as the alternative pathway of complement activation.
发表于 2025-3-22 19:24:55 | 显示全部楼层
A Brief History of Inflammatione phenomenon was recognized by Galen (3rd century, A.D.) as a reaction of the body against injury, as well as by . (1794), an English doctor of the 18th century. An interesting wealth of historical information about that period can be found in the series of papers by . (1970–1972).
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The Release of Hydrolytic Enzymes From Phagocytic and Other Cells Participating in Acute and Chronicner towards an extracellular stimulus. Also viable cells under certain conditions, can be induced to release newly synthesized hydrolases over a long period of time, illustrating a possible mechanism by which tissue damage, degradation and remodelling seen during chronic inflammatory processes can occur.
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