Titlebook: Infectious Diseases from Nature: Mechanisms of Viral Emergence and Persistence; C. J. Peters,Charles H. Calisher Conference proceedings 20

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under our noses and no one notices,α, and IgG2a antibodies in immunity..Human monocytotropic ehrlichiosis (HME) manifests as a flu-like illness that progresses in severity to resemble toxic shock-like syndrome, with meningoencephalitis or adult respiratory distress syndrome in some patients, and requires hospitalization in half. In i

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Population dynamics of RNA viruses: the essential contribution of mutant spectra,y. The great adaptive potential of viruses, conferred on them by high mutation rates and quasispecies dynamics, demands new strategies for viral disease prevention and control. This necessitates a more detailed knowledge of viral population structure and dynamics. Here we review studies with the imp

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Control of arbovirus diseases: is the vector the weak link?,ceptable health and economic burden on humans and animals, including livestock. The developed world has been fortunate to have escaped much of the burden that arboviruses and their arthropod vectors inflict on humans in disease endemic countries, but the introduction and rapid spread of West Nile vi

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,Pathogenesis of Rift Valley fever virus in mosquitoes — tracheal conduits & the basal lamina as an mosquito ingests viremic vertebrate blood, virus infects midgut epithelial cells and replicates, then disseminates to other tissues, including salivary glands and/or ovaries. The virus is then transmitted to the next vertebrate host horizontally via bite and/or vertically to the mosquito’s offspring

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The virulence of the 1918 pandemic influenza virus: unraveling the enigma,tissue of 1918 influenza victims, the complete genomic sequence of the 1918 influenza virus is being deduced. Neither the 1918 hemagglutinin nor neuraminidase genes possess mutations known to increase tissue tropicity that account for virulence of other influenza virus strains, such as A/WSN/33 or t