Titlebook: Heart rate as a determinant of cardiac function; Basic mechanisms and G. Hasenfuss,H. Just Conference proceedings 2000 Steinkopff Verlag Da

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J. Ross Jr. M.D.t principles, as shown briefly in the following sections. This in turn permits a broad extrapolation towards the unknown and relatively complex mixtures and blends of a wide variety of commercial and other combustible compounds.

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https://doi.org/10.1007/978-3-642-47070-7cardiac function; drugs; force-frequency relation; heart; heart failure; heart rate; hemodynamics; myocardi

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re of great relevance for the prognosis of cardiac patients. This book provides a summary of the current knowledge on the influence of heart rate on myocardial function and hemodynamics in non-failing and failing animal and human hearts. The subcellular and molecular alterations underlying the alter

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R. Haque,H. Kan,M. S. Finkelctions. These discussions cover the significance of the results obtained from relevant standard test procedures. The procedures themselves, together with the background philosophy, are covered in appendix 3.

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Influence of SR Ca2+-ATPase and Na+-Ca2+-exchanger on the force-frequency relationolic function may result from reduced SR Ca. ATPase and is most pronounced in failing hearts with lack of upregulation of the Na.-Ca. exchanger. Thus, failing hearts with reduced SR Ca. ATPase protein levels and unchanged Na.-Ca. exchanger protein levels exhibit severe impairment of both systolic and diastolic function.

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Force-frequency relation in patients with left ventricular hypertrophy and failurejection fractions well above 75%. Clinical symptoms include angina-like chest discomfort, orthostatic intolerance, and exertional dyspnea. Both the chest pain and dyspnea have been linked to subendomyocardial hypoperfusion with worsened diastolic failure (2, 4, 5, 15, 16, 38).

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Physiology and pathophysiology of baroreceptor function and neuro-hormonal abnormalities in heart fae experimental evidence for an abnormality in the function of the arterial- and cardiopulmonary baroreceptor reflexes and 3) discuss, whether this abnormality or an interaction with renal mechanisms might cause the neuro-hormonal derangements in congestive heart failure.

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