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,Transforming growth factor-ß induction in carcinoid heart disease,d by their ability to produce and secrete a variety of bioactive substances, e.g. serotonin, histamin, prostaglandins, kallikrein and tachykinins. These or related substances produce the clinical symptoms of flush, diarrhea and tachycardia as well as bronchospasm, cyanosis and edema.

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Fibroblast and transforming growth factors in the heart: A role in cardiac growth ?,f cardiac adaptive growth is the function, if any, of peptide growth factors in the cardiac myocyte. The following article will examine the involvement of two key growth factors in relation to the cardiac myocyte.

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Growth factor signal transduction in the cardiac myocyte: functions of the serum response element,cal or pharmacological models of hypertrophy [17–20]— there have been surprisingly few direct attempts to test the promising hypothesis that growth factors might modulate or govern critical aspects of the cardiac phenotype. One regrettable truism, which often is cited to help account for this paucit

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Growth factors and development of coronary collaterals,4]. In canine heart, epicardial vessels increase from an initial diameter of 40 .m to an average diameter of 80 .m [1,4,5,31]. Hence, it can be stated that progressively slow coronary artery stenosis favours the induction of blood vessel growth, a phenomenon called angiogenesis. Angiogenesis in the

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