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The role of growth factors in angiogenesis,occurred over the last 5 years. Widespread use of recombinant DNA technology has led to the identification of new peptide growth factors (and in many cases their receptors) which are capable of stimulating angiogenesis. For example, the family of Heparin Binding Growth Factors (HBGF’s), which includ

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Atherosclerosis and platelet derived growth factors, 65 years of age in the Western world. The economic impact of CHD is correspondingly profound, with estimated costs for treating the condition in England and Wales alone exceeding £400 million per annum [2]. Our understanding of the cellular events that lead to atherosclerosis have improved substant

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,Effects of TGF-ß on vascular smooth muscle cell growth,e if any growth and express smooth muscle-specific contractile proteins which enable them to maintain vascular tone. However, abnormal VSMC growth occurs in a number of vascular disease processes. In hypertension the artery wall becomes thickened as a result of growth (probably hypertrophy) of contr

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Some aspects of growth signal transduction in vascular smooth muscle cells,luminal pressure such as occurs in arteriovenous fistulas and in pulmonary hypertension. The intimai smooth muscle accumulation in atherosclerosis, restenosis after angioplasty and in vein grafts is arguably a consequence of these same repair and haemodynamic adaptation mechanisms [1]. Intimai VSMC

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