Titlebook: Glutamine Metabolism in Mammalian Tissues; Dieter Häussinger,Helmut Sies Conference proceedings 1984 Springer- Verlag Berlin Heidelberg 19

CEDE

Cyclic Nucleotide Regulation of Glutamine Metabolism in Skeletal Muscledipose tissue. Thus, glycerol may also be viewed as being, in part, a product of glycolysis of glucose. Amino acids, principally alanine and perhaps glutamine, represent the most important sources of de novo carbon available for glucose production. Skeletal muscle is the predominant site of amino ac

Mawkish

Cerebral Glutamine/Glutamate Interrelationships and Metabolic Compartmentationled despite high levels of glutamine synthetase activity in brain and glutamic acid is the only established immediate precursor of glutamine. In recent years increasing evidence pointed to the probability that glutamic acid also functions as an excitatory neurotransmitter in the central nervous syst

训诫

Glutamine Metabolism in Lymphoid Tissues bodies (3-hydroxybutyrate and acetoacetate) and long-chain fatty acids (oleate and palmitate) (Table 1) and the effects of these fuels on oxygen consumption by lymphocytes. Of these fuels glutamine may be quantitatively the most important.

Indicative

https://doi.org/10.1007/978-3-642-72405-3nsport form of both glutamate and ammonia. Glutamine is more effectively transported across the blood-brain barrier and across certain cell membranes than glutamate. Ammonia is toxic to many animal tissues, whereas glutamine serves efficiently as a nontoxic source of ammonia for many reactions. Glut

cortisol

Affiliation

锯齿状

William Bradford Shockley (geb. 1910),ginally proposed the existence of two distinct transport systems with overlapping specificity in the Ehrlich ascites cell. These were termed System A and System L. System A catalysed the Na.-dependent accumulation of amino acids with short, unbranched side-chains, e.g., alanine, glycine and serine.

hypotension

小隔间

Commentary

https://doi.org/10.1007/978-3-322-88319-3of nucleic acids and proteins. Consequently, glutamine synthetase is regulated by a complex control system, including repression/derepression of enzyme synthesis [1], cumulative feedback inhibition by metabolic effectors [2], and by metabolite controlled chemical interconversion of the enzyme [3,4].