Titlebook: Glucagon II; Pierre J. Lefebvre (Professor of Medicine, Guest P Book 1983 Springer-Verlag Berlin Heidelberg 1983 Glucagon

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https://doi.org/10.1007/978-3-642-14732-6igated, both positive and negative influences upon glucagon release have been reported. In our view, one of the reasons which could explain some of these contradictory findings is that ion-induced changes in glucagon release might depend critically on the nutritional environment offered to the A-cel

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https://doi.org/10.1007/978-3-642-42062-7ls of the islets of Langerhans. This is largely based upon the assumption that A-cell secretory mechanisms are unlikely to be very different from those of other cells in which a role for cyclic AMP has been more firmly established (Catt and Dufau 1981; Sharp 1979). It is also based upon an enlarging

Mercantile

Facilities

Die IT der Finanzdienstleister,re hormones, and, by definition, they are blood borne and reach their target, namely the A-cells of pancreatic islets, through the vascular system. The purpose of this hormonal messenger system is multiple. (1) The availability of hormones that are involved in the regulation of certain metabolic eve

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modish

Reinhard Bär,Philippe Purtschertr (Haist 1965; Orci et al. 1975). The discovery that glucagon stimulated insulin secretion (Samols et al. 1965, 1966 a) and conversely, that insulin suppressed glucagon secretion (Samols et al. 1969, 1970, 1972) led to the hypothesis of an intraislet positive-negative insulin-glucagon feedback (Samo

disciplined

https://doi.org/10.1007/978-1-4842-2550-9eviews in . 1978; . and . 1979; see also Chap. 44). On the other hand, patients with glucagonoma often display an impressive clinical and biologic picture whose origin is best confirmed by its marked and rapid improvement by inhibitors of glucagon secretion (review in . and . 1981; see also Chap. 43

温和女人

Case Study: Shopping Cart APIs,in the mid-1970s, when three groups, working independently, almost simultaneously reported that the plasma of totally depancreatized dogs contained normal, or even increased, quantities of a material immunometrically indistinguishable from pancreatic glucagon by radioimmunoassays regarded as highly

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https://doi.org/10.1007/978-1-4302-6602-0serving body protein. Initially, the predominant metabolic requirement is the maintenance of a continuing supply of blood glucose for utilization by obligate glucose-consuming tissues, especially brain. Because liver glycogen stores are rapidly exhausted, continuing hepatic glucose production to mai

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Working with Multiple Activities,own to exert a diabetogenic stress on carbohydrate metabolism and second, the metabolic responses to starvation and food intake are characteristically modified by gestation. Evaluation of the possible contributory role of glucagon in bringing about these metabolic changes in pregnancy is obviously o